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GABA is released, primarily via Ca 2+ -dependent conventional exocytosis and Ca 2+ -independent mechanisms via GABA transporters [201], from GABAergic horizontal cells, amacrine cells, to BPCs and RGCs, where GABA is crucial for the processing of visual information and the development of retinal circuitry in the inner retina [202]

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These studies focus on understanding biological processes rather than demonstrating clinical effectiveness