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The endothelial cells were selectively expanded by switching to endothelial cell (EC) media supplemented with retinoic acid (RA) (D7-D8)

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Although many scientists acknowledge that the lack of carcinogenicity of DEHP in humans is primarily based on indirect evidence and peroxisome proliferation cannot be definitively identified as the sole mechanism of DEHP carcinogenicity, the possibility of DEHP tumorigenesis via non-PPARα pathways, such as nuclear factor kappa B (NFκB), androstane receptor (CAR), and pregnant X receptor (PXR), remains unclear