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Epileptic astrocytes are also characterized by partial loss of glutamate synthetase and reduced GABA availability 317 , increased expression of astrocyte-specific adenosine kinase—which limits adenosine levels and further decreases adenosine A1 receptor-mediated inhibition 318 —loss of AQP4 polarization at astrocytic endfeet 319 and downregulation of astrocytic Cx43

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It catalyses hydroperoxides and protects biomembranes and cell structures from oxidative damage