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The reduction in serum UA levels by rasburicase, a recombinant urate oxidase that decreases circulating UA ∼90% [9], led to a decrease in serum and saliva antioxidant capacity, as assessed by the total radical-trapping antioxidant potential (TRAP) and ferric-reducing antioxidant potential (FRAP) assays

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[96] confirmed that HPF, due to its physico-chemical properties, is easily incorporated into the membrane lipid bilayer, where it can function as a protonophore, triggering proton crossing regardless the involvement of any channel protein