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Acute intense exercise can induce immunosuppression through Arg1 upregulation, whereas long-term, regular, moderate-intensity exercise tends to shape an anti-inflammatory environment [113], improving the overall immune regulatory capacity and T-cell function through mechanisms such as optimizing the Arg1/iNOS balance and reducing myeloid-derived suppressor cell (MDSC) suppressive effects—particularly important for maintaining T-cell function during aging [114]

dinitrobenzene (CDNB) conjugation with reduced glutathione (1.0 mM) in 100 mM KPO4 (pH 6.5) at room temperature.