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When the Δψ m is high, like in cancer cells, the electron transfer is slowed down, as protons have to be pumped against an enhanced electrochemical force, so that an electron leakage and, consequently, high ROS production are favored [47] (Figure 1)

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Additionally, certain pharmacological treatments for COPD, particularly systemic corticosteroids, are known to exacerbate hyperglycaemia and increase the risk of developing T2D, further complicating disease management, while antidiabetic drugs, such as glucagon-like peptide-1 receptor agonists (GLP-1RAs) and sodium–glucose cotransporter-2 inhibitors (SGLT-2is), may exert beneficial effects beyond glycaemic control, potentially improving pulmonary outcomes and systemic inflammation in patients with COPD [7,8,9,10,11,12]