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For instance, angiotensin II infusion-upregulated NOXs and mitochondrial oxidative damage, followed by significant cardiac hypertrophy, fibrosis, and diastolic dysfunction, may be improved through the administration of mitochondrial-targeted anti-oxidant agents [137]

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“Insights Into the Role of Tetrahydrobiopterin Deficiency in the Pathogenesis of Gestational Hypertension.” Hypertension , vol